Adrenocorticotropin evokes transient elevations in intracellular free calcium ([Ca2+]i) and increases basal [Ca 2+]i in resting chondrocytes through a phospholipase C-dependent mechanism

Jodi F. Evans; Chwan-L Shen; Simcha Pollack; John F. Aloia; James K. Yeh

doi:10.1210/en.2004-1612

Adrenocorticotropin evokes transient elevations in intracellular free calcium ([Ca²⁺]_i) and increases basal [Ca ²⁺]_i in resting chondrocytes through a phospholipase C-dependent mechanism

Jodi F. Evans
, Chwan-L Shen
, Simcha Pollack
, John F. Aloia
, James K. Yeh

Biology, Chemistry, Environmental Studies

Texas Tech University Health Sciences Center
Winthrop University Hospital

Research & Scholarship: Contribution to journal › Article › peer-review

Original language	English
Pages (from-to)	3123-3132
Number of pages	10
Journal	Endocrinology
Volume	146
Issue number	7
DOIs	https://doi.org/10.1210/en.2004-1612
State	Published - Jul 2005

ASJC Scopus Subject Areas

Endocrinology

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Adrenocorticotropin evokes transient elevations in intracellular free calcium ([Ca²⁺]_i) and increases basal [Ca ²⁺]_i in resting chondrocytes through a phospholipase C-dependent mechanism. / Evans, Jodi F.; Shen, Chwan-L; Pollack, Simcha et al.
In: Endocrinology, Vol. 146, No. 7, 07.2005, p. 3123-3132.

Research & Scholarship: Contribution to journal › Article › peer-review

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title = "Adrenocorticotropin evokes transient elevations in intracellular free calcium ([Ca2+]i) and increases basal [Ca 2+]i in resting chondrocytes through a phospholipase C-dependent mechanism",

author = "Evans, \{Jodi F.\} and Chwan-L Shen and Simcha Pollack and Aloia, \{John F.\} and Yeh, \{James K.\}",

note = "Both clinical and in vitro evidence points to the involvement of the melanocortin peptide, ACTH, in the terminal differentiation of chondrocytes. Terminal differentiation along the endochondral pathway is responsible for linear growth, but also plays a role in osteoarthritic cartilage degeneration.",

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AU - Pollack, Simcha

AU - Aloia, John F.

AU - Yeh, James K.

N1 - Both clinical and in vitro evidence points to the involvement of the melanocortin peptide, ACTH, in the terminal differentiation of chondrocytes. Terminal differentiation along the endochondral pathway is responsible for linear growth, but also plays a role in osteoarthritic cartilage degeneration.

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